By K. D. Rainsford (auth.), Gy. Mózsik, L. Nagy, A. Pár, K. D. Rainsford (eds.)
The clinical major programmes of those sequence of symposia replaced now and then following the overseas clinical developments. the most programmes of the 1st Symposium have been: 1. Gastric and intestinal cytoprotection; 2. Hepatoprotection; three. Pancreatic safety. For the second: 1. Gastrointestinal mucosal 'cytoprotection'; 2. Liver 'cytoprotection'; three. Pancreatic 'cytoprotection'; four. unfastened radicals and scavangers; five. laptop method of cytoprotec tion. For the 3rd one: 1. easy, critical, peripheral and mobile mechanisms of gastrointestinaol cytoprotection; 2. Esophagal safety; three. Gastric safeguard; four. Small intestinal damage and defense; five. huge bowel damage and defense; 6. Liver damage and defense; 7. Pancreas damage and defense. the most clinical programmes of the Fourth foreign Symposium on 'Cell harm and safety within the Gastrointestinal Tract' are: 1. basic mechanisms of gastrointestinal harm and safety; 2. phone harm and security within the belly; three. mobile harm and security within the small gut and within the huge bowel; four. telephone damage and defense within the liver and pancreas; five. mobilephone damage and safety of the premalignant prestige and malignant illnesses within the gastrointestinal tract. The offered papers are released during this booklet. The abstracts of this assembly have been released in Digestive ailments and Sciences, and we list appreciation of the Editor ofthatjoumal for his or her book. many of the papers released right here have seemed in Inflammopharmacology 1996;4:331-398.
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Extra resources for Cell Injury and Protection in the Gastrointestinal Tract: From Basic Sciences to Clinical Perspectives 1996
KIRALY, G. stJTO, Gy. RUMI, I. SZABO AND A. VINCZE First Department of Medicine, Medical University of Pees, Ifjusag ut, H-7643 Pees, Hungary *Correspondence This paper was first published in: Inflammopharmacology. 1996;4:361-378. ABSTRACT Prostacyclin (PGh) and ~-carotene have a key role in gastric mucosal defence against endogenous or exogenous noxious agents. Prostacyclin has appreciable protective effects on the gastrointestinal (GI) mucosa, while ~-carotene (as one of the retinoid compounds) has oxyradical scavenging properties.
O - ';; ~*:~ . -*t. ----, o 10 20 30 40 50 60 Time after HCI administration (min) Figure 15. PGIrinduced (5 and 50 Ilg/kg ig) and ~-carotene-induced (I and 10 mg/kg ig) changes in the gastric mucosal adenylate pool (ATP+ADP+AMP) of HCI-treated rats in relation to dose and time after HCl administration. See Figure I for further explanation 44 Mozsiket al. 0 /I-carotene - v - 5 I-Ig/kg .. - I ................... --4~~~------------. ---,---, o 10 20 30 40 50 60 v 15 2 "- . - - o +----r--~----~---r--~----, o 10 20 30 40 50 60 Time after ETOH administration (min) Time after ETOH administration (min) Figure 12. PGIrinduced (5 and 50 Ilg/kg ig) and p-carotene-induced (l and 10 mg/kg ig) changes in the gastric mucosal AMP of EtOH-treated rats in relation to dose and time after EtOH administration. •• - \ "If c: 'v .. I '2 "~ "If _. ~~ .. , a.. ::::; + I Time after Hel administration (min) I I.
I ................... --4~~~------------. ---,---, o 10 20 30 40 50 60 v 15 2 "- . - - o +----r--~----~---r--~----, o 10 20 30 40 50 60 Time after ETOH administration (min) Time after ETOH administration (min) Figure 12. PGIrinduced (5 and 50 Ilg/kg ig) and p-carotene-induced (l and 10 mg/kg ig) changes in the gastric mucosal AMP of EtOH-treated rats in relation to dose and time after EtOH administration. •• - \ "If c: 'v .. I '2 "~ "If _. ~~ .. , a.. ::::; + I Time after Hel administration (min) I I.